GASTROINTESTINAL AND LIVER DISEASE – Exercise 43

The multiple choice questions in this online test paper focuses on GASTROINTESTINAL AND LIVER DISEASE MCQ. If you are looking for self evaluation of your PG Medical entrance exam preparation then this online GASTROINTESTINAL AND LIVER DISEASE MCQ Test Paper will help you to evaluate your exam preparation.

Instruction for REPRODUCTIVE BIOLOGY/ REPRODUCTIVE MEDICINE / INFERTILITY MCQs:

1- This online MCQ practice test paper contains 30 questions.
2- Each question in this online practice test paper have four options and only 1 option is correct.
3- You can view the answers of this practice test paper after submitting the practice test paper.
Note: The answers mentioned at the end of practice test are the best suitable option as per our knowledge. Users shall cross-check the answers with their textbooks.

Q 1. false statement regarding hepatorenal syndrome

urinalysis normal

renal biopsy is normal

kidneys can be used for renal transplantation

hypernatremia

Q 2. mercaptans are derived from intestinal metabolism of

threonine

methionine

leucine

isoleucine

Q 3. true regarding hepatorenal syndrome

splanchnic vasodilation

arteriovenous shunting

profound renal vasoconstriction

all of the above

Q 4. first clotting factor to be depleted in cirrhosis liver is

factor V

factor VII

factor VIII

factor IX

Q 5. hepatorenal syndrome is treated with

midorine

octreotide

intravenous albumin

all of the above

Q 6. true for type 2 hepatorenal syndrome

most have refractory ascites

increase in serum creatinine is moderate

no tendency of serum creatinine to prograss over time

all of the above

Q 7. typical smell in fetor hepaticus is due to

mercaptans

ammonia

bilirubin

all of the above

Q 8. false statement regarding hepatorenal syndrome

treatment is usually unsuccessful

vasodilator therapy with intravenous infusions of low dose dopamine is effective

TIPS can improve renal function

treatment of choice is liver transplantation

Q 9. in patients of advanced cirrhosis or acute liver failure hepatorenal syndrome occurs in

~5%

~10%

~20%

~30%

Q 10. type 2 hepatorenal syndrome is mainly characterized by

hepatic encephalopathy

refractory ascites

hypotension

all of the above

Q 11. following has a role in the treatment of hepatic encephalopathy

azithromycin

ritonavir

rifamixin

lumefantrine

Q 12. goal of lactulose therapy is to promote how many soft stools per day

2-3

4-6

6-8

8-10

Q 13. type I hepatorenal syndrome is characterized by doubling of serum creatinine level to >2.5 mg/dl in

< 2 weeks

< 4 weeks

< 6 weeks

< 8 weeks

Q 14. disorders that can mimic clinical features of hepatic encephalopathy are all except

meningitis

hypoglycemia

hypocalcemia

Wilsons disease

Q 15. false regarding hepatic encephalopathy

disturbances in consciousness

behavior and personality changes

fluctuating neurologic signs

no electroencephalographic changes

Q 16. true for type I hepatorenal syndrome

progressive oliguria

rapid rise of serum creatinine

common precipitating event in spontaneous bacterial peritonitis

all of the above

Q 17. disorders that can mimic clinical features of hepatic encephalopathy are all except

wernickes encephalopathy

korsakoffs psychosis

subdural hematoma

schizophrenia

Q 18. neurological signs in hepatic encephalopathy includes all except

rigidity

decreased deep tendon reflexes

extensor plantar signs

seizures

Q 19. true regarding hepatorenal syndrome

type I HRS is the more aggressive form

type I HRS carries a mortality rate of >90%

HRS is seen in patients with refractory ascites

all of the above

Q 20. true regarding lactulose

nonabsorbable

disaccharide

leads to colonic acidification

all of the above

Q 21. supplementation recommended in patients with hepatic encephalopathy

copper

zinc

calcium

magnesium

Q 22. false regarding hepatic encephalopathy

reduced levels of consciousness is due to excessive concentration of GABA in the CNS

endogenous benzodiazepines act through the GABA receptor

1,4 benzodiazepines is isolated from brain tissue of patients with fulminant hepatic failure

excessive magnesium deposition in basal ganglia contribute its pathogenesis

Q 23. false statement regarding hepatorenal syndrome

worsening azotemia

avid sodium retention

oliguria without identifiable causes of renal dysfunction

kidneys are structurally smaller

Q 24. in hepatic cirrhosis which clotting factor is not reduced

factor II

factor VII

factor XI

Q 25. disorders that can mimic clinical features of hepatic encephalopathy are all except

acute alcohol intoxication

sedative overdose

delirium tremens

encephalitis

Q 26. false regarding hepatic encephalopathy

blood brain barrier is intact

ammonia is incriminated in its pathogenesis

many patients have elevated blood ammonia levels

mercaptans , short chain fatty acids & phenol are incriminated in its pathogenesis

Q 27. false statements about spontaneous bacterial peritonitis is

therapy is for 10 to 14 days

prophylactic norfloxacin reduces recurrences

primary prevention recommended in high risk cases

empirical coverage for anaerobes is necessary

Q 28. true regarding hepatic encephalopathy

more common in chronic liver disease

essential for diagnosis of fulminant hepatic failure

diagnosis of hepatic encephalopathy is clinical

all of the above

Q 29. most common predisposing factor for hepatic encephalopathy

gastrointestinal bleeding

increased dietary protein

electrolyte disturbances

injudicious use of CNS depressing drugs

Q 30. earliest sign of hepatic encephalopathy is

EEG changes

astrexis

reversal of sleep/wake cycle

deterioration in handwriting

GASTROINTESTINAL AND LIVER DISEASE – Exercise 43

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GASTROINTESTINAL AND LIVER DISEASE – Exercise 43

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